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Description
This process yields an excess of reactive oxygen species (ROS), which impairs endothelial function, reduces the bioavailability of nitric oxide (NO), and triggers the release of tissue factor (TF), cumulatively contributing to the formation of microclots in the pulmonary vasculature [3,4]

Lei et al., 2025 for reviews)

& Biswal, S

doi: 10.1007/s00213-022-06285-4
3582 - 3588, Journal articles | 2014 Zong X
