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Clinically, strategies aimed at modulating EAAC1-mediated cysteine transport and boosting endogenous GSH synthesis may hold promise for mitigating post-ischemic neurodegeneration and cognitive deficits associated with vascular dementia

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The three differentially altered pathways between the LC vs HCs were glutamine, nitrogen, and butanoate mechanisms whereas cysteine and methionine, taurine, and glutathione metabolisms were altered between the LC and R groups ( Figure 4A )