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Cerebral ischemia-reperfusion therapy aims to mitigate or reverse brain injury but may paradoxically induce oxidative stress, inflammation and apoptosis, thereby exacerbating cerebral damage (189)

For certain inhibitors (e.g., 32 and 50 ), an increase in absorbance signal was also observed at 10 M

Keaney and Campbell, 2015)

Endotoxin contamination a common source of confounding in peptide research can trigger inflammatory responses in biological assays that mimic or mask the compounds actual biological activity

In vitro characterization and inhibition of the interaction between ciprofloxacin and berberine against multidrug-resistant Klebsiella pneumoniae