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In summary, we introduce a programmed targeting nanosystem based on a sequentially responsive liposome to dual rectify the abnormal energy and stromal microenvironments via coordinated regulation of GLUT1 and NF-B signal in PDAC cells and PSC, thus improving the anti-cancer efficacy of PDAC

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Full therapeutic doses are a separate and more promising story, now in trials for depression and, as covered above, in an early test in Parkinsons

Circ-MBOAT2 knockdown represses tumor progression and glutamine catabolism by miR-433-3p/GOT1 axis in pancreatic cancer