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Although rupture of the plasma membrane represents the terminal event of ferroptotic death, emerging evidence indicates that ferroptotic signaling is initiated at intracellular membrane compartments, including the ER, Golgi-associated vesicles, and lysosomes 107-110

Secondly, NBDHEX has been found to dissociate the TRAF2-GSTP1 complex in human osteosarcoma cells (U-2OS), therefore inducing activation of JNK and p38 downstream signals and eventually apoptosis [11]

Its primary job is to combat oxidative stress by neutralizing harmful free radicals unstable molecules that can damage cells, proteins, and DNA