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Vascular smooth muscle cells specific deletion of angiopoietin-like protein 8 prevents angiotensin II-promoted hypertension and cardiovascular hypertrophy

Furthermore, GSH can modulate protein function via S-glutathionylation under oxidative stress

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Additionally, mitochondrial calcium ions modulate key enzymes of the tricarboxylic acid cycle, such as pyruvate dehydrogenase, thereby regulating OXPHOS (13)

Considering the putative role of oxidative stress as a pathogenetic factor for the development of colitis and because the reductive metabolism of TNBS has been shown to generate ROS, this model of colitis was chosen to evalute the temporal relationship between the levels of GSH in mucosa and the appearance and extent of injury in vivo during the acute phase of colitis in TNBS+ethanol-treated rats and to determine the effect of NAC administration on mucosal GSH levels and extent of injury