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doi: 10.1016/j.addr.2018.03.009

Additionally, high levels of ROS can cause DNA damage and hence higher frequencies of mutation (Figure 1)
Edaravone and benidipine protect myocardial damage by regulating mitochondrial stress, apoptosis signalling and cardiac biomarkers against doxorubicin-induced cardiotoxicity

Structure-activity relationship work has produced a series of analogues built around the ipamorelin scaffold
This review comprehensively explores the recent advances in understanding the multifaceted role of oxidative stress in sepsis pathophysiology, with particular emphasis on mitochondrial dysfunction and the emerging therapeutic potential of antioxidants to restore redox balance, improve organ function, and enhance patient outcomes
