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The possible underlying mechanism is associated with enhanced hepatic lipid oxidation via an Adenosine Monophosphate- Activated Protein Kinase- Acetyl-CoA carboxylase (AMPKAcetyl-CoA carboxylase) pathway, which leads to a reduction in liver fatty acid synthase activity, an increase in glycogen storage, and a decrease in tumor necrosis factor expression [157]

C.LuoX.ArakiS.et al (2011)

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Improvement of hepatic and extrahepatic functions and anti-inflammatory effects of nitazoxanide in disease models of LPS-induced systemic inflammation and acute-on-chronic liver failure, in The international liver congress, cirrhosis and complications (London, UK: European Association for the Study of the Liver EASL)

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