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Nevertheless, further specific studies are needed to clearly assess the role of CBS polymorphism in atherosclerotic disease, particularly in its interactions with folate deficiency, vitamin B 6 deficiency, and methionine intake

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Additionally, oxidative stress promotes further ROS production and influences the activity of cysteine-dependent proteases [30]

This mitochondrial dysfunction may lead to arrest of cell division, triggered by OS [15, 16]

Activation of the G protein leads to the release of the alpha subunit, which then activates an effector enzyme or ion channel