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Reducing iron toxicity accumulation and mitochondrial dysfunction through CISD1 gene inhibition enhances the process of erastin-induced iron ptosis, which does not involve GPx4 [43]

The optimal concentration (30 g/ml) was therefore selected for follow-up studies that showed an enhanced rate of neuronal differentiation, dendrite complexity, axonal development and synaptogenesis, with all responses consistent with the hormetic dose response

Maes K, Serre J, Mathyssen C, Janssens W, Gayan-Ramirez G

14 Becerril-OrtegaJ.BordjiK.FreretT.RushT.BuissonA

Severe inflammatory ophthalmopathy in a euthyroid patient during nivolumab treatment