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Both priming and activation are blocked by treatment with genistein, a tyrosine kinase inhibitor, suggesting an important role for tyrosine kinase signaling pathway in activating NADPH oxidase (McLeish et al., 1998

IAC, the reaction product of TEMPO and presumably also TEMPOL, showed protective effects in animal models of various diseases, including colitis, Alzheimers disease, and diabetes (Novelli et al., 2007

[DOI] [PMC free article] [PubMed] [Google Scholar] Broad RC, Bonneau JP, Hellens RP, Johnson AAT

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