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[DOI] [PMC free article] [PubMed] [Google Scholar] 131

Adolph TE, Mayr L, Grabherr F, Schwarzler J, Tilg H

Results Neuronal GCLC knockout leads to brain atrophy accompanied by neuronal cell death To examine how the declines in GCLC and glutathione might affect brain homeostasis, we prepared GCLC floxed X CaMKII-Cre (GCLC-KO) mice whose GCLC expression is deleted in calcium/calmodulin-dependent protein kinase II (CaMKII)-positive neurons

In humans, a primary source of ROS is the metabolic byproduct end of the respiratory chain located in the inner mitochondrial membrane

In mice with intestinal dysbiosis, KCs induce NKT cell overactivation, leading to increased IFN- levels through enhanced IL-12 expression, ultimately inhibiting effective liver regeneration after PHx