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In conclusion, our study demonstrated that CD38 deficiency significantly alleviated Ang II-induced hypertension and vascular remodeling by inhibiting the SA-sEV-mediated senescence of VSMCs

The peptide's unique molecular mechanisms involving angiogenesis pathway activation, growth hormone receptor upregulation, and anti-inflammatory modulation distinguish it from conventional therapeutic approaches and provide biological rationale for its observed effects in experimental models

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At the metabolic level, AOD 9604 promotes triglyceride hydrolysis while concurrently reducing lipogenic activity through modulation of enzymes such as acetyl-CoA carboxylase

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