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FAK localization to focal adhesions (FAs) is regulated through the interaction between the FAK FAT domain and key adapter protein, paxillin, whereas FAK kinase activity does not affect FAK localization 22,38

These mechanisms involve different steps which are currently not well-characterized: (1) entrance of Ca 2+ into the enterocytes through the Ca 2+ -selective transient receptor potential (TRP) channel TRPV6 and the voltage-gated calcium channel Cav1.3, a member of the L-type calcium channel family