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HEP-1 also activated the PI3K-AKT signaling pathway, increased the expression of endothelial nitric oxide synthase (eNOS), promoted the expression of serum and glucocorticoid-induced kinase (SGK), and enhanced the biosynthesis of epidermal growth factor (EGF) and vascular endothelial growth factor (VEGF), thereby facilitating the repair and healing of gastric mucosal injury (GMI)

Scanning electron microscopy analysis reveals that hairs from GHK-Cu-treated models show more regular cuticle structure, increased cortical density, and improved overall fiber integrity

These findings further improved the function of FGF5 gene, and provided therapeutic ideas for androgen alopecia.

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