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Figures Abstract We have previously reported that acetylsalicylic acid (aspirin, ASA) induces cell cycle arrest, oxidative stress and mitochondrial dysfunction in HepG2 cells

The absence of serious adverse events, treatment-related withdrawals, IGF-1 elevation, glucose metabolism impairment, or immunogenic responses represents a favorable safety foundation

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The bulk RNA sequencing data generated in this study have been deposited in the Gene Expression Omnibus (GEO) for Mouse data (derived from C57BL/6 wild-type organoids): GSE310410 and Human organoid data: GSE310408

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