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This phosphorylates and inhibits GFPT1, thereby weakening YAP protein stability and the transcription of its downstream target gene FTH1, ultimately promoting ferritinophagy, lipid peroxidation, and enhanced ferroptosis susceptibility (50)

Scientists are actively researching several potential mechanisms underpinning CVS, including: Neurotransmitter Imbalances: The condition may result from hyperexcitability in the nervous system, leading to hyperemesis

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10.1016/j.jhep.2020.06.036 15