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Description
Targeting just one pathway produces significant results

These findings suggest potential relevance not just for longevity, but also for regenerative medicine and age-related tissue repair

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From a mechanistic point of view, Cu@HER2@NPs limit the growth, migration, and invasion of U251 glioblastoma via HER2 receptor-mediated endocytosis and trigger DNA damage-mediated p53 signaling pathways

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