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Tumor necrosis factor-alpha (TNF-) serves as a principal mediator of inflammation and is chiefly governed by the transcription factor nuclear factor-kappa B (NF-B)

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Because of the high heterogeneity of tumors, according to Darwinian evolutionary principles, drug-resistant mutants are selected, or subpopulations of primary resistant tumor stem cells in the dormant phase can cause tumors to regrow or spread

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