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To be able to relate the presence of this signature to the exposure to electrochemically silent toxic metals can strengthen the hypothesis of its role in a general mechanism based on induction of a condition of chronic oxidative stress- the somatic and carcinogenic effects of toxic metals and of other disease-generating conditions

Both of these processes are mainly regulated by the signaling pathway: insulin-like growth factor-1 (IGF-1) phosphoinositide-3-kinase (PI3K) protein kinase B (Akt) mammalian target of rapamycin (mTOR)

Imaging the substantia nigra in parkinson disease and other parkinsonian syndromes

Furthermore we observed a concomitant increase in cells undergoing early apoptosis, as shown by cells expressing annexin V only (p = 0.0023), and cells staining for both PI and annexin V (p = 0.0218) ( Figure S2B )