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IFX inhibited the expression of HDAC6 and HDAC7 which are associated with inflammation and cartilage degradation, while enhancing HDAC4 expression, which negatively regulates Runx2 , decreased the activation of NF-B, Jun N-terminal kinase (JNK), and ERK

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According to the findings, copper chelators and copper transporter protein inhibitors significantly reduced neuronal cell mortality, ameliorated neurocognitive deficits, and averted A deposition [83, 84]