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An improved understanding of genetic forms of disease has more recently facilitated the identification of amenable targets for novel gene therapies in subsets of patients with ALS

However, there is an alternative mechanism for proteasomal degradation of Nrf2 independent of Keap1, which is mediated by glycogen synthase kinase 3 (GSK3) and -TrCP (Figure 1B)

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The diagnosis of shoulder separation is often quite apparent from hearing a story that is typical of this injury, followed by a simple physical examination