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Aberrant N6-methyladenosine (m6A) modification of GPX4 mRNA contributes significantly to epigenetic tumorigenesis, and here, we report that selective methyltransferase-like 3 (METTL3)-dependent m6A methylation of GPX4 plays a key role in C5AR1 knockdown-induced ferroptosis induction

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It plays a central role in tissue regeneration, wound healing, collagen synthesis, and anti-aging processes, and is naturally present in human plasma and extracellular matrix

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