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These experimental studies allow researchers to explore the biochemical mechanisms that regulate hormone signalling and intracellular regulatory responses

[DOI] [PMC free article] [PubMed] [Google Scholar] 202.Tomasi M.L., Cossu C., Spissu Y., Floris A., Ryoo M., Iglesias-Ara A., Wang Q., Pandol S.J., Bhowmick N.A., Seki E., et al

These findings confirm that COP1 acts as a key mediator of TLR4/NF-B activation, and miR-146b-enriched EVs exert therapeutic effects via COP1 suppression

doi: 10.1016/j.beem.2013.05.003

Moreover, Wang et al., used a maternal immune activation (MIA) mouse model to examine whether probiotic supplementation during pregnancy can prevent autism-related phenotypes in offspring