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Science 342(6156):373377
Kyn stimulated mTOR in DN T cells (Fig
New England Journal of Medicine , 379 (22), 21402150
Hemoglobin's oxygen-binding capacity is decreased in the presence of carbon monoxide because both gases compete for the same binding sites on hemoglobin
This paradigm positions mtDNA heteroplasmy not merely as a passive biomarker of dysfunction, but as a programmable regulator of tumor behavior, with broad implications for targeting the LARS2 mitochondrial translationS100A8/A9 axis in cancer therapy (Fig
