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Free radicals are harmful atoms that can damage DNA, causing many illnesses associated with premature ageing
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The rationale rests more on theoretical benefit and observed clinical patterns than controlled trial evidence
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Preclinical studies in in vitro models have shown that simvastatin exerted its anti-tubercular activity through cholesterol-driven autophagy, which is mediated by the adenosine monophosphate-activated protein kinase (AMPK)mammalian target of the rapamycin complex 1 (mTORC1)TFEB axis [58] (Fig
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Frontiers in Pharmacology, 2017, 8: 904
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