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Particular focus should be placed on interactions between mHTT, the ironmetabolism axis (such as transferrin receptor 1 and hepcidin), and key ferroptosis mediators (GPX4 and Nrf2) to build a dynamic model linking mHTT aggregation, iron dysregulation, and neuronal ferroptosis, and to define subtype-specific vulnerability of MSNs

Stem cell-based tissue replacement after stroke: factual necessity or notorious fiction

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10.1039/c5bm00161g 22 CaoY.da Silva AraujoM.LorangC
