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Nevertheless, the lack of IL-17 exacerbates DSS-induced colitis, suggesting that IL-17 also has a beneficial effect (Strober et al., 2002)

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[1] , which assessed the relationship between homocysteine metabolism and the histological severity of nonalcoholic fatty liver disease (NAFLD), and modeled how replacement with different cofactors, such as pyridoxine (vitamin B6), cobalamin (vitamin B12), betaine, and folic acid (vitamin B9), may affect hepatic homocysteine levels in patients with NAFLD