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SOCS proteins bind JAKs or their receptors and act by (i) abolishing the kinase ability of JAK, (ii) abolishing the ability of JAK to bind to the receptor, (iii) blocking STATs from being recruited to the receptor, and (iv) mediating the ubiquitination of JAKs and STATs for degradation (124)

Indeed, as one study found, thymalin reduced the expression of CD44 and CD117 by 2-3 times and increased the expression of CD28 by 6.8 times

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Consistent with this observation, patients with IGF1 gene deficiency are growth retarded (321)