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Jacobs received financial support from the Struwig-Germeshuysen Kankernavorsingstrust

doi: 10.14740/jocmr2815w

Restoration of DJ-1 expression ameliorates these defects by enhancing mitochondrial electron transport chain activity and reducing ROS overproduction, thereby attenuating oxidative stress-mediated neuronal damage (McCoy and Cookson, 2011)

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