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Inability of volatile anesthetics to inhibit oxygen-glucose deprivation-induced glutamate release via glutamate transporters and anion channels in rat corticostriatal slices

In this context, NF-B activation plays a major role in mediating inflammation, as the p65 subunit is released from its inhibitor IB (inhibitor of kappa B), translocates to the nucleus, and promotes pro-inflammatory cytokine gene expression [14]