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Skipping of exon 14 removes Y1003, a key phosphorylation site required for CBL-mediated c-MET ubiquitination and subsequent degradation, resulting in aberrant c-MET accumulation and prolonged signaling, thereby driving unchecked cellular proliferation [45,46,47,48]

This may be caused by hyperinsulinaemia during the onset of the disease

For those comfortable with injection protocols, this route provides higher bioavailability and lower cost but requires more effort and risk assessment