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Description
They found that 62 percent had lactase deficiency, 16 percent were sucrase-deficient, and 10 percent were maltase-deficient [12]
Most (70%) of these mutations are missense mutations affecting conserved residues in structurally and functionally important domains of PK
Future research should focus on elucidating L-carnitine's molecular mechanisms within the hepatic metabolic network and rigorously assessing its safety and efficacy in well-designed clinical trials
Not intended to diagnose, treat, cure, or prevent any condition
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