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Antioxidant enzyme deficiencies, for dyslipidemic patients, were: 35% for sinus bradycardia and associated arrhythmias, 33% for sinus tachycardia, 33% for atrial fibrillation, 32% for atrial flutter and atrial extrasystolic arrhythmia, 30% for extrasystolic and ventricular arrhythmia, and 26% for paroxysmal supraventricular tachycardia

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To better understand the efficacy and safety of cell replacement therapy, a variety of experimental models of PD have been established

Mitochondrial dysfunction can contribute to mitochondrial DNA (mtDNA) mutations, increased mitochondrial reactive oxygen species (mtROS), and oxidative stress, which have been progressively underscored in the pathogenesis of RA over recent years (9)

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