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[PubMed] [Google Scholar] Scian M., Atkins W.M

10.1016/j.ejca.2009.12.008 35 CebullaN.SchirmerD.RunauE.FlammL.GommersbachS.StengelH.et al (2023)

The subsequent series of adverse events include loss of membrane integrity, mitochondrial dysfunction, impaired sperm motility as well as DNA damage and apoptosis resulting in damage of cellular components and pathogenesis of reproductive diseases and infertility ( Thus, information obtained from the assessment of OS status could be of great importance in enhancing clinical decision making

GSH From Antioxidant to Post-Translational Modifier (PMC, 2016) S-Glutathionylation as a Major Redox Signalling Mechanism A review published in PMC, authored by a leading researcher in the field, documented GSHs evolution in the scientific literature from a simple antioxidant to a key post-translational modifier through S-glutathionylation the reversible formation of mixed disulfides between GSH and protein cysteine residues