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Description
B.MiuraT
Oxidative stress activates NUPR1 by both ER stress (METH [79] and H2O2 [80]) and non-ER stress pathways, NUPR1 regulates ferroptosis via iron metabolism, ROS homeostasis, and the GSH/GPX4 pathway [81, 82]
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The ischemic gut generates a substantial amount of ROS, which, along with a surge of inflammatory mediators (e.g., TNF-, IL-6), enters the circulation
