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Prenatal stress and risk for autism

To determine the mechanism by which oxidative stress promotes HO-1 expression, we showed that tBHP or CHP promoted the degradation of Bach1, which was blocked by pretreatment with the proteasome inhibitor MG-132 (Fig

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Corver WE, Demmers J, Oosting J, Sahraeian S, Boot A, Ruano D, et al
