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Such alterations were also associated with inhibition of cytochrome c oxidase (CcOX) activity and microtubule network disassembly, which are considered hallmarks of nitric oxide (NO) toxicity, indicating that NO, rather than the depletion of GSH per se, is the primary mediator of cell damage
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Lomustine further decreased protein expression of the programmed cell death markers cleaved PARP, cleaved caspase 3, phospho-RIPK3, and MLKL (Figure 7F)
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Characterization of the anticonvulsant properties of ganaxolone (CCD 1042
