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In addition, an increasing amount of research has supported a pathophysiological role for ferroptosis in the development of cardiovascular diseases, including myocardial ischemiareperfusion injury, myocardial infarction, adriamycin-mediated myocardial injury, and heart failure [69]

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By contrast, ACSL3 transforms monounsaturated fatty acids into acyl-CoA esters, which bind to membrane phospholipids, thereby preventing ferroptosis in cancer cells