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The gut-organ axis: clinical aspects and immune mechanisms
doi: 10.1016/j.resp.2020.103455 102 RodriguesT
2f, g), a concern raised in a previous study 52
These are all evidence of compensatory glutamatergic activity 31,43,44,45 and indicate that initial disruption of the balance between excitatory glutamatergic and modulatory dopaminergic neurotransmission is sufficient to disrupt normal synaptic plasticity
Hydrogen peroxide as a signaling molecule
