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The C-terminal difference between the two variants makes GAC more specifically active, which may contribute to its greater susceptibility to overexpression in aggressive cancers (Redis et al., 2016)
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However, the downstream mechanisms by which CDK4/6 inhibitors exert their antitumor effects in HCC remain largely unexplored
doi:10.1146/annurev-nutr-120524-043056 Quinn M, Halsey J, Sherliker P, et al
Amyloid-Beta Peptide Disruption of Lipid Membranes and the Effect of Metal Ions
