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Ligand-dependent genomic function of glucocorticoid receptor in triple-negative breast cancer

On the other hand, cancer cells sometimes exploit a reversible inactivation of GAPDH via oxidative modification of its active-site cysteine (a GAPDH redox switch), which diverts glucose flux into the pentose phosphate pathway to generate NADPH for antioxidant defense 114

Expression of peripheral blood lymphocyte muscarinic cholinergic receptor subtypes in airway hyperresponsiveness

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