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As a consequence, pharmacological inhibition of TRAP1 has been proposed as an anti-neoplastic approach for MPNST and other tumor types

Resistance mechanisms, such as GPX4 upregulation, further complicate treatment

Silybin can counteract this by activating the deacetylase SIRT2 (likely through increasing intracellular NAD+ levels via downregulating PARP-1 expression)

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Moreover, exposure of neutrophils to Bacillus proteins resulted in further neutrophil activation via inositol 1,4,5-triphosphate, and elevated production of IL-1 and IL-6 [58]