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This regulatory mechanism underscores DJ-1s crucial role in cellular defense against oxidative stress, highlighting its potential as a therapeutic target for neurodegenerative diseases and conditions involving oxidative damage (Wang H

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Neuroscience 95, 425432

Ku70 -/- MEFs were generated by introduction of Cas9-sgRNA targeting Xrcc6 intron 4 and exon 6 (Supplementary Table 1) and were complemented by re-expression of Ku70 (+KU70) using lentiviral transduction (Supplementary Fig

ACR neuroinflammation is driven primarily by oxidative stress, inflammation, and disturbances in neurotransmitter function