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In contrast, treatment with DL-propargylglycine (PAG), a CSE inhibitor, exacerbated T2DM-induced liver injury, leading to severe hepatocellular vacuolation, heightened oxidative stress, and amplified inflammation [76]

Taken together, these data reveal that IAV- infected DC significantly increase glycolysis without increased shuttling of acetyl CoA from pyruvate into the TCA cycle, while also improving their capacity and flexibility for mitochondrial substrate oxidation, including markedly increasing glutaminolysis

The mechanisms of cellular uptake were explored by addition of free targeting ligands, endocytic and metabolic inhibitors, co-localization of nanocarriers with intracellular organs, and surface charge modification of cells

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