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Illum, L., Jrgensen, H., Bisgaard, H., Krogsgaard, O
This review will focus on the underlying pathophysiological and molecular mechanisms linking GSH and its precursors with T2D and OxS
In this model, trigonelline normalized levels of SOD, catalase, glutathione, and nitric oxide synthase activities [197]
Bellini, M., et al
doi: 10.1021/acschembio.7b00730
