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Abstract Background Cyclophosphamide (CP)-induced hepatotoxicity is a significant problem in clinical settings

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MGF has been shown to stimulate muscle stem cells (satellite cells) to re-enter cell cycle and proliferate, resulting in new muscle cells to replace injured fibers [10, 12]

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A sufficient treatment response was achieved in both treatment groups

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From gut to liver Exploring the crosstalk between gut-liver axis and oxidative stress in metabolic dysfunction-associated steatotic liver disease

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Energy is lower

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