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This facilitates K48-linked ubiquitination of OCTN2, accelerating its degradation via the autophagy-lysosome and endocytosis-lysosome pathways [104, 105]

Unfortunately, even though patients tolerated treatment well and most adverse events were mild and reversible, there was only one partial response among 19 evaluable patients with glioblastoma and no response among 6 patients with anaplastic glioma [105]

Reduction of S-nitrosoglutathione by alcohol dehydrogenase 3 is facilitated by substrate alcohols via direct cofactor recycling and leads to GSH-controlled formation of glutathione transferase inhibitors

The systemic effects of AKI that may contribute to mortality have been extensively investigated in preclinical models

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