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Activation of ERK is dependent on the RAS/MAPK pathway, in which IIS results in phosphorylated IRS or SHC that recruit growth factor receptor-bound protein 2 (GRB-2)
Gupton-Campolongo T
Heterogeneity of growth hormone-dependent lipolytic factors
High ROS induce NF-B signaling activation, up-regulate inflammatory cytokines (TNF-, IL-1) which promote inflammation, and further induce the abnormal expression of VEGF, induce abnormal angiogenesis (74)
