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Mechanistically, Ahrr deficiency results in ferroptosis in Ahrr/ IELs through the AHR-induced expression of CYP1A1, a monooxygenase that generates ROS, increasing oxidative stress, redox imbalance and lipid peroxidation (65)

Understanding RAGE, the receptor for advanced glycation end products

National Toxicology Program, U.S

It guides RISC to cleave the target sequence, thus achieving therapeutic effects by downregulating the translation of specific proteins
